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Giving people back their lives!

Harch Hyperbarics and The Family Physicians’ Center Congratulate Our HBOT Lottery Winners

Harch Hyperbarics and The Family Physicians’ Center
Congratulate Our HBOT Lottery Winners




Shany, Lara, and Tyler

To all of the lovely people who have written in expressing their desire to try this life changing therapy, we would like to say:
  • Never give up hope
  • You are on the right track by researching Hyperbaric Oxygen Therapy (HBOT). We urge you to continue to learn about HBOT, and write to your congressmen and senators. Stress them to advocate for insurance reimbursement for hyperbaric oxygen therapy for neurological disorders and traumatic brain injuries (TBI). 
  • If you did not win, we still have your story and you will be entered in the next lottery for another chance to win.
At Harch Hyperbarics and Family Physicians' Center we believe in doing everything we can to help the patients.

Hyperbaric Oxygen Therapy in Emergency Medicine

HBOT in Emergency Medicine

K. Van Meter, L. Weiss, and P.G. Harch


As an entry from K. K. Jain's Textbook Of Hyperbaric Medicine all chapter references refer to the 4th Edition.
Hypoxemia and ischemia are the underlying pathologies in many of the conditions seen in an emergency department. In addition to resuscitation and other emergency treatments, hyperbaric oxygen plays a vital role in the management of these patients. This topic is discussed under the following headings:
Timely resuscitation by augmentation of oxygen delivery to tissue damaged by ischemia is key to many emergency medicine interventions in sickness and injury. Further, the prompt attempt to lessen reperfusion injury and necrosis after initial resuscitative clinical success should not be forgotten. Finally, the patient, once past the initial resuscitative effort, followed by restorative oxygenation, should receive maintenance oxygenation as needed to optimize the chance of continued recovery. In other words, one of the major purposes of an emergency department is to first assure proper oxygen delivery to many of its sick and injured. The oxygen delivery must be adjusted to maximize therapeutic effect in the emergency medicine interventional phases of resuscitation and restoration, and the maintenance phase of patient management.
Oxygen delivery to tissue is dependent on cardiac output. Oxygen delivery to tissue is given by the following formula:
Oxygen delivery = cardiac output x arterial oxygen content 
(Shannon &Celli 1991)

Hyperbaric Oxygen Therapy in the Management of Cerebral Palsy

HBOT in the Management of Cerebral Palsy

Virginia Neubauer, Richard Neubauer and Paul Harch


An entry from K.K. Jain's Textbook Of Hyperbaric Medicine

Cerebral palsy is a chronic neurological disorder that can be due to several causes of brain damage in utero, in the perinatal period, or postnatally. Hyperbaric oxygen has been shown to be useful in treating children with cerebral palsy. This topic is discussed under following headings:
Causes of Cerebral Palsy
Oxygen Therapy in the Neonatal Period
Treatment of Cerebral Palsy with HBOT


Causes of Cerebral Palsy

The term cerebral palsy (CP) covers a group of non-progressive, but often changing, motor impairment syndromes secondary to lesions or anomalies of the brain arising in the early stages of development. Between 20 to 25 of every 10,000 live-born children in the Western world have the condition (Stanley et al 2000). Problems may occur in utero, perinatal, and postnatal. Infections, traumatic brain injury, near-drowning and strokes in children suffering from neurological problems come under the heading of cerebral palsy. Diagnosis of cerebral palsy resulting from in utero or early perinatal causes may be made immediately after birth, but more commonly occurs between 15 and 24 months. It is possible that CP may be misdiagnosed for years because specific symptoms may show up very late in childhood. Some of the possible causes of Cerebral Palsy and are listed in Table 21.1.
Although several antepartum causes have been described for CP, the role of intrapartum asphyxia in neonatal encephalopathy and seizures in term infants is not clear. There is no evidence that brain damage occurs before birth. A study using brain MRI or post-mortem examination was conducted in 351 full-term infants with neonatal encephalopathy, early seizures, or both to distinguish between lesions acquired antenatally and those that developed in the intrapartum and early postpartum period (Cowan et al 2003). Infants with major congenital malformations or obvious chromosomal disorders were excluded. Brain images showed evidence of an acute insult without established injury or atrophy in (80%) of infants with neonatal encephalopathy and evidence of perinatal asphyxia. Although the results cannot exclude the possibility that antenatal or genetic factors might predispose some infants to perinatal brain injury, the data strongly suggest that events in the immediate perinatal period are most important in neonatal brain injury. These findings are important from management point of view as HBOT therapy in the perinatal period may be of value in preventing the evolution of cerebral palsy.

We Welcome Our Patients to Harch Hyperbarics and the Family Physicians' Center

Harch Hyperbarics and
The Family Physicians' Center


Approximately 90% of our patients fly in from out of town.

We do what we can to assist in making arrangements for their comfort while they are here in New Orleans. 
The need has arisen to create a SERVICE where certified Nurses’ Aides would be available to assist in the care of out of town patients.
Our Nurses’ Aides are available to help take care of your loved one in a hospital setting, Long Term Care (LTC) Facility, Rehab, or Ronald McDonald House.
 Our Nurses’ Aides work closely with an RN who mentors them and is available on call 24 hours a day, 7 days a week.

For more information, just ask Juliette at the private office.

(504) 309-4948
Veterans Call Toll Free 1-(855)-438-4268




Hyperbaric Oxygen Theraphy in Global Cerebral Ischemia/Anoxia and Coma

HBOT Therapy in Global Cerebral Ischemia Anoxia and Coma

Paul G. Harch and Richard A. Neubauer

 An entry from K.K. Jain's Textbook Of Hyperbaric Medicine

Hyperbaric oxygen therapy has been used in a number of conditions characterized by global ischemia (as opposed to focal ischemia of stroke), and anoxia, and leading to impairment of consciousness. Conditions such as coma due to brain injury and anoxia associated with drowning and hanging are discussed under the following headings:



For a discussion of the effectiveness of hyperbaric oxygen (HBOT) therapy in global cerebral ischemia/anoxia and coma, we define HBOT as a medical treatment that uses high pressure oxygen as a drug by fully enclosing a person or animal in a pressure vessel and then adjusting the dose of the drug to treat pathophysiologic processes of the diseases. Like all drugs, the dose of HBOT is crucial and should be customized to each patient's response. It is dictated by the pathological target and is determined by the pressure of oxygen, duration of exposure, frequency, total number of treatments, and timing of the dose in the course of the disease. As diseases and their pathologies evolve, different doses of HBOT are required at different times. In addition, patients have individual susceptibility to drugs, manifest side effects and toxicity. Unfortunately, the ideal dose of HBOT in acute or chronic global ischemia/anoxia and coma is unknown. The studies reviewed below suggest higher pressures (2 ATA or higher) and lesser numbers of treatments very early in the disease process whereas lower pressures (2 ATA or lower) and a greater number of treatments have been used as the brain injury matures. While this general trend seems justified, the absolute or effective pressures delivered to the patients in these reports may be slightly less than what is stated since many studies do not specify the HBOT delivery system that was employed. For example, an oxygen pressurized chamber has an effective HBOT pressure equal to the plateau pressure administered during the treatment, whereas an air pressurized chamber in which oxygen is administered by aviators mask can achieve a far lower effective HBOT pressure, depending on the fit of the mask and the amount of its air/oxygen leak. In the later cases, the dose of oxygen is less. This concept is particularly important when analyzing the studies in this chapter performed prior to the late 1980s when the aviator mask dominated delivery systems in multiplace chambers.

Tommy Tarlton Races for a Cause to Benefit Wounded Warriors


Please view John Salcedo's movie Brain Storm "Thunder Bowl" at  RealShowInt.Com

Join the Cause this Spring 2011

VTX Wounded Worrier Bike Run


 California VTX Riders Honor a Wounded Warrior at the 8th Annual Wounded Warrior Run

Please join us on this day as we honor this years Wounded Warrior. Our event is a fundraiser and 100% of funds collected will be donated to our Warrior. Join us for lunch, drinks, live music and dancing, visiting with old friends and making new ones. It's for the Warrior so please join us.

Our day starts at Loma Linda VA Hospital in Riverside, CA to have breakfast with our Veterans. Then we move to the junction of Hwy 138 and Interstate 15 to line up for the ride up the hill at 10:00am, we will stop at Silverwood lake to take a group picture then on to the BBQ!!

There will be T-shirts for sale and well some very good food! All proceeds are donated to our Wounded Warrior. You do not need a motorcycle to join us, and this is a family event!! See you there!!

IHMA - Report from Inside the Beltway, Washington, D.C.

Compressed air began being used to treat Caisson's disease and diving injuries over 120 years ago. Back in 1936, 74 years ago, Behnke published the diving tables using oxygen instead of just compressed air to treat diving injuries. Oxygen diving tables were not adopted by the U.S. Navy until 1968!

Unfortunately, Hyperbaric oxygen therapy has never been taught in U.S. medical schools, and there are only a few Hyperbaric medicine fellowships. Until recently, in 2003 when the IHMA petitioned Medicare and got diabetic foot wounds approved because it prevents 75% of all amputations, Hyperbaric medicine was only available at about 500 locations. Now it is available at over 1,000 locations and increasing weekly. With it, continuing medical education is being pursued by physicians, and many more physicians today are aware of the benefits of saturating the body's tissues with oxygen. Thanks to Congressman Istook's efforts, Hyperbaric oxygen therapy is again receiving grants from the National Institutes of Health, and Dr. Stephen Thom has published some ground-breaking research as a result. The previous testimony to Congress, before Dr. Harch's in 2002, was in 1963. I assure you that no other medical specialty has taken so long to tell Congress what they are able to do.

Unfortunately, though DoD developed this medicine, it has not been fully implemented to the level of known benefit to patients. This is largely because everyone "thought" they were treating bubbles as the cause of decompression sickness, the primary indication. It turned out they were generally treating damage the bubbles cause, not just bubbles. That was not discovered until 1989. Many today still believe they are treating bubbles, and this misunderstanding has held back the science and acceptance of HBOT as a therapy for general application to the repair of underlying biological processes common to many conditions.

CRAO a Continuing Look at HBOT as a Treatment for this Vascular Event

We believe our experience with HBOT for CRAO justifies continuing its use as the treatment of choice. It is hoped that our experience will encourage more referrals in the favorable first eight hours. The intensive treatment regimen was satisfying to us, and we hope others find it equally effective in preserving vision.  HBOT is a logical treatment for central retinal artery occlusion.  This continuing study corroborates improvement of vision using HBOT when the occlusion is under 24 hours, and further study will be needed to determine effectiveness of treatment after a 24-hour period has elapsed.